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Mitochondrial ROS production and oxidative mtDNA damage is increased during reoxygenation in the GPX1 knockout mice, in addition to structural abnormalities in cardiac mitochondria and myocytes, suggesting GPX1 may play an important role in protecting cardiac mitochondria from reoxygenation damage in vivo

In conclusion, our study provides a robust methodology for redox profiling and establishes baseline data for NAD and glutathione metabolites in healthy populations

It constitutes a source of reducing equivalents to scavenge ROS [44,45]

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Gaussian 09, Revision A.08, Frisch, M