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Given the strong association between steatotic liver disease and cardiometabolic disorders [10] , this research suggests the possibility of linking deranged copper metabolism with metabolic dysfunction-associated steatotic liver disease (MASLD)

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Once -arrestin is bound to a GPCR, it undergoes a conformational change allowing it to serve as a scaffolding protein for an adaptor complex termed AP-2, which in turn recruits another protein called clathrin

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