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It was first isolated from bovine muscle in 1905 by two Russian scientists [24], and only the L-isomer was found to be bioactive [4]

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Biochemical and histological analyses were performed on the hippocampal and cortical tissue, assessing oxidative stress markers, antioxidant enzyme activity, and glial activation through spectrophotometric assays and immunohistochemistry

10.1016/j.msard.2020.102312 96 MedinaL

HFD-induced obese mice increased AF vulnerability and exhibited apparent atrial structural remodeling, including left atrial dilation, cardiomyocyte hypertrophy, connexin-43 remodeling and fibrosis