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The intrinsic pathway involves mitochondrial permeability transition pore (MPTP) opening, controlled by BCL-2 family proteins (e.g., BAX/BAK)

Coupled with the absence of clinical outcome measures and human clinical trial evidence, their direct clinical translation is restricted, highlighting an urgent need for more clinically relevant studies to optimize intervention strategies and clarify the actual therapeutic value of iron chelators in DR patients

Fibrates may cause an abnormal urinary betaine loss which is associated with elevations in plasma homocysteine

Selvarajah, D