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Cinobufotalin exerts its anticancer effects through various mechanisms, such as inhibiting cell proliferation and colony formation and blocking cell cycle-related pathways [206]

1E), indicating that these MIM-dependent l-EVs, rather than general l-EVs, have the ability to transfer Rac1 to promote cell migration

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MicroRNA-21 (miR-21) represses tumor suppressor PTEN and promotes growth and invasion in non-small cell lung cancer (NSCLC)