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R.SunK.et al

Loss of metabolic plasticity underlies metformin toxicity in aged Caenorhabditis elegans

The glucose metabolic reprogramming in CRC centers on the EMP-TCA cycle nexus, glucose uptake is facilitated initiated by glucose transporter 1 (GLUT1)-mediated glucose influx [13]

(2017), via metabolomics by 1D 1H Nuclear magnetic resonance (NMR) along with immunologic evaluations, showed substantial overlap between the metabolic profile driving macrophage polarization either into the M1 phenotype or the M2 phenotype and showed that the P

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