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The peptide also shows benefits in models of peripheral nerve injury, where topical application enhances nerve regeneration rates by 35% and improves functional recovery scores by 40%

190 One study further explored the downstream consequences of this co-localization and demonstrated for the first time that cytoplasmic CTSB, rather than trypsin, initiates acinar cell death in AP

52 Indeed, various experimental epilepsy models have reported that the overproduction of ROS triggers an increase in inflammation in neurons

VipA interferes with organelle trafficking by acting as a nucleator for actin polymerization[14]

The short half-life (~30 min) allows GH to be released in natural pulses rather than sustained elevation, preserving physiologic rhythm