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We show that UNC1 functions noncell autonomously, in neurons, and disruption of the gene led to secretion of an excess of a sulphated signal that activates NHR1 in different tissues

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doi: 10.3109/15513815.2012.659410 27 PontesFSCde SouzaLLde AbreuMCFernandesLARodriguesALMdo NascimentoDMet al

Additionally, it has been established as a therapeutic option for some neurological disorders such as Parkinsons disease, Alzheimers disease, and multiple sclerosis (3840)