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Mice with a deficiency of Prx2, the more abundant peroxiredoxin in RBCs, have decreased hematocrit levels and an increased amount of circulating reticulocytes (106)

Other work has implicated Notch signaling (specifically, DLL4-Notch1) in arteriolar responsiveness and capillary pericyte status

In line with the energy starvation hypothesis [30], which induced that energy supply scarcity was responsible for the contractile dysfunction in heart failure