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Some agents have proposed cell cycle and apoptosis pathway effects (Fig

Vaamonde-Garcia, C

Collectively, these results indicate that preventing C-terminal lysine modifications of GLT-1 reduces neuronal injury following ischemic insult, supporting a role for GLT-1 ubiquitination as a mechanism driving aberrant transporter trafficking and excitotoxic damage

Under specific inflammatory conditions, Tregs can transdifferentiate into Th17-like cells, characterized by the production of pro-inflammatory cytokines such as IL-17, thereby promoting disease progression in autoimmune and inflammatory disorders (68, 70)

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