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Wild-type TP53 hampers the shunt of the glucose carbon to anabolic pathways by binding to and inhibiting the G6PD, whereas mutant TP53 is unable to affect the G6PD activity (175)

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The protein expression levels of ischemic and non-ischemic gastrocnemius muscle tissues at different time points (the 2nd, 5th, 7th, and 14th day post-HLI) were determined by western blot analysis (Fig
The use of prescription stimulant medication as a maintenance therapy for MUD has received a corresponding groundswell of support from researchers and clinicians [14]