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NNSMS contributed to designing the outlines of the literature study

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They next established that mitochondrial Coenzyme Q counteracted ferroptosis triggered by pharmacological and genetic inhibition of GPX4, suggesting that mitochondrial lipid redox signaling regulates ferroptosis in AML cells

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Through its copper-binding properties, GHK-Cu may contribute to antioxidant defense systems that protect tissues from oxidative stress and free radical damage