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LPS remains the most widely adopted stimulus to trigger a robust pro-inflammatory (M1-like) phenotype, either alone or in combination with cytokines such as IFN- or TNF, which further potentiate downstream signaling through NF-B, Janus kinase/signal transducer and activator of transcription (JAK/STAT), mitogen-activated protein kinase (MAPK), and inflammasome pathways ( Supplementary Table 1 ) (231258)

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