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Recently, our approach was the BPC 157 therapy effect (for review see, i.e., [ 2 4 ]) in the study of the permanent occlusion of major vessels-induced occlusion syndromes [ 14 20 ], the occlusion-like syndrome induced by severe intoxication (alcohol, lithium) [ 21 , 22 ] or maintained severe intra-abdominal hypertension [ 23 ] recovered with the activation of the collateral pathways [ 14 23 ]

We began with the presumption that the reducing potential of cytoplasmic thiol redox components was required to prevent disulfide bonds from forming

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Never self-administer or exceed the recommended dose