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Consistent with the model proposed by Datsi and Garg, TIL plus PD-1 strategies aim to increase both the number and functional quality of tumor-specific T cells beyond a critical threshold, rather than merely releasing inhibitory brakes on an insufficient endogenous repertoire [24]

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Our previous work in human macrophages indicates that HIV blocks the formation of the apoptosome and that in surviving cells Bim, a highly apoptotic protein, is recruited to the mitochondria without resulting in apoptosis 23 , suggesting that in HIV surviving cells, mitochondrial function is compromised

Co-Chairs: Dominique Forrester, Federal Facilities Chief, California Department of Toxic Substances Control

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