GSH serves as a substrate for glutathione peroxidases and glutathione S-transferases and participates in reversible protein S-glutathionylation (Lu, 2013)
Sciatic nerve surgery for the functional evaluation cohort was performed using the same protocol as above stated except that no neuronal tracing was executed, and the rats were sacrificed 6 months post-injury by transcardial perfusion with saline followed by buffered 4% paraformaldehyde
The amplitude activity of both the first and second neurons rose significantly
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