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doi: 10.1210/endocr/bqab202, 204

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Furthermore, iron deficiency disrupts mitochondrial function and its essential role in oxidative metabolism, ATP production (via the tricarboxylic acid cycle), and related enzymatic activities, leading to dysregulated cellular metabolism

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However, if one component of the redox buffer partitions selectively into the MLO, that would lead to a very different redox environment in the MLO compared to its environment