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We showed that RSPO2-deficient mice have reduced active -catenin signalling and a decreased mineral deposition rate and osteoblast number, similar to the phenotype observed with the knockout of LRP5/6

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This eventually leads to a reduced capacity in the brain to think clearly and sustain attention

Inside this molecule, the incretin arms counterbalance that effect, and the net result in trials was strong glucose control plus an extra energy-expenditure component

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